Alzheimer DiseaseRobert D. Terry Lippincott Williams & Wilkins, 1999 - Broj stranica: 457 The Second Edition of this highly acclaimed volume examines every major aspect of Alzheimer disease -- clinical, epidemiologic, structural, chemical, genetic, molecular, and therapeutic. This edition includes expanded coverage of the cellular-level exploration of related dementing disorders, with in-depth presentation of prion diseases, Pick's disease, fronto-temporal disorders, transgenic models, and biochemistry of presenilins. Treatment of symptoms with therapeutic drugs is discussed, along with outcomes of and problems inherent in clinical trials for AD. The broad coverage of AD in this book benefits clinicians, educators, investigators, and health administrators. |
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Stranica 282
... neuronal loss has also been reported in the dorsal raphe nucleus ( 0–39 % ) and the central superior nucleus ( 0–36 % ) ( 176–181 ) . The loss of serotonin- ergic neurons seems to be on average considerably smaller than the reported loss ...
... neuronal loss has also been reported in the dorsal raphe nucleus ( 0–39 % ) and the central superior nucleus ( 0–36 % ) ( 176–181 ) . The loss of serotonin- ergic neurons seems to be on average considerably smaller than the reported loss ...
Stranica 373
... loss ( 1-4 ) . A number of brain regions are particularly affected by the loss of neurons , including the hippocampus and the entorhinal cortex ( 4,5 ) . D. A. Peterson : Department of ... Neuronal Death Daniel A Peterson and Fred H Gage.
... loss ( 1-4 ) . A number of brain regions are particularly affected by the loss of neurons , including the hippocampus and the entorhinal cortex ( 4,5 ) . D. A. Peterson : Department of ... Neuronal Death Daniel A Peterson and Fred H Gage.
Stranica 452
Robert D. Terry. Neurofibrillary tangles ( contd . ) formation , neuronal types prone to , 216-217 laminar distribution , 210 and plaques density , and loss of cholinergic innervation , 280 and loss of cholinergic neurons , 280-281 ...
Robert D. Terry. Neurofibrillary tangles ( contd . ) formation , neuronal types prone to , 216-217 laminar distribution , 210 and plaques density , and loss of cholinergic innervation , 280 and loss of cholinergic neurons , 280-281 ...
Sadržaj
Clinical Presentation | 6 |
Noncognitive Symptoms of Alzheimer Disease | 25 |
Neuropsychology of Alzheimer Disease | 39 |
Autorska prava | |
Broj ostalih dijelova koji nisu prikazani: 26
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abnormal Acad Sci USA activity allele Alzheimer disease Alzheimer's disease amyloid precursor protein Ann Neurol ApoE apolipoprotein apoptosis Arch Neurol areas Aẞ associated atrophy B-amyloid basal forebrain behavioral Biol Brain Res cells cerebral cortex cholinergic cholinergic neurons chromosome clinical cognitive impairment correlation cortical deficits degeneration demented density diagnosis disorders drug effects elderly entorhinal ethnic Exp Neurol familial frontal frontotemporal dementia function gene genetic growth factor hippocampus human immunoreactivity increased lesions levels Lewy body lobe Masliah memory mer's disease metabolism microglia monkeys mutations Natl Acad Sci neocortex neuritic Neurobiol Aging neurodegenerative neurofibrillary tangles neurofilament Neurology neuronal loss neurons neuropsychological Neurosci Neurosci Lett normal onset Parkinson disease pathological patients peptide phosphorylation presenilin prevalence Proc Natl Acad Prpsc Psychiatry receptor regions reported risk factors senile dementia senile plaques studies subcortical symptoms synaptic syndrome tau protein temporal tion transgenic mice trophic factors vascular dementia