Alzheimer DiseaseRobert D. Terry Lippincott Williams & Wilkins, 1999 - Broj stranica: 457 The Second Edition of this highly acclaimed volume examines every major aspect of Alzheimer disease -- clinical, epidemiologic, structural, chemical, genetic, molecular, and therapeutic. This edition includes expanded coverage of the cellular-level exploration of related dementing disorders, with in-depth presentation of prion diseases, Pick's disease, fronto-temporal disorders, transgenic models, and biochemistry of presenilins. Treatment of symptoms with therapeutic drugs is discussed, along with outcomes of and problems inherent in clinical trials for AD. The broad coverage of AD in this book benefits clinicians, educators, investigators, and health administrators. |
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Stranica 14
... present with cognitive complaints of uncertain significance or demonstrate only questionable deficits on quantitative test performance . Particularly when an infor- mant's report is lacking to address the issue of functional ...
... present with cognitive complaints of uncertain significance or demonstrate only questionable deficits on quantitative test performance . Particularly when an infor- mant's report is lacking to address the issue of functional ...
Stranica 153
... present with a mixed form of dementia . Moreover , the conceptual basis of these criteria is flawed : the utility of amalgamating numerous diseases , with such diverse pathophysiological mechanisms , based solely on the fact that they ...
... present with a mixed form of dementia . Moreover , the conceptual basis of these criteria is flawed : the utility of amalgamating numerous diseases , with such diverse pathophysiological mechanisms , based solely on the fact that they ...
Stranica 331
... present in all brain regions , with the strongest labeling in neurons and the neuropil , including axons and dendrites ; weaker immunoreactivity was present in glial cells . Subcel- lular fractionation of monkey cortex revealed PS1 ...
... present in all brain regions , with the strongest labeling in neurons and the neuropil , including axons and dendrites ; weaker immunoreactivity was present in glial cells . Subcel- lular fractionation of monkey cortex revealed PS1 ...
Sadržaj
Clinical Presentation | 6 |
Noncognitive Symptoms of Alzheimer Disease | 25 |
Neuropsychology of Alzheimer Disease | 39 |
Autorska prava | |
Broj ostalih dijelova koji nisu prikazani: 26
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Uobičajeni izrazi i fraze
abnormal Acad Sci USA activity allele Alzheimer disease Alzheimer's disease amyloid precursor protein Ann Neurol ApoE apolipoprotein apoptosis Arch Neurol areas Aẞ associated atrophy B-amyloid basal forebrain behavioral Biol Brain Res cells cerebral cortex cholinergic cholinergic neurons chromosome clinical cognitive impairment correlation cortical deficits degeneration demented density diagnosis disorders drug effects elderly entorhinal ethnic Exp Neurol familial frontal frontotemporal dementia function gene genetic growth factor hippocampus human immunoreactivity increased lesions levels Lewy body lobe Masliah memory mer's disease metabolism microglia monkeys mutations Natl Acad Sci neocortex neuritic Neurobiol Aging neurodegenerative neurofibrillary tangles neurofilament Neurology neuronal loss neurons neuropsychological Neurosci Neurosci Lett normal onset Parkinson disease pathological patients peptide phosphorylation presenilin prevalence Proc Natl Acad Prpsc Psychiatry receptor regions reported risk factors senile dementia senile plaques studies subcortical symptoms synaptic syndrome tau protein temporal tion transgenic mice trophic factors vascular dementia