Alzheimer DiseaseRobert D. Terry Lippincott Williams & Wilkins, 1999 - Broj stranica: 457 The Second Edition of this highly acclaimed volume examines every major aspect of Alzheimer disease -- clinical, epidemiologic, structural, chemical, genetic, molecular, and therapeutic. This edition includes expanded coverage of the cellular-level exploration of related dementing disorders, with in-depth presentation of prion diseases, Pick's disease, fronto-temporal disorders, transgenic models, and biochemistry of presenilins. Treatment of symptoms with therapeutic drugs is discussed, along with outcomes of and problems inherent in clinical trials for AD. The broad coverage of AD in this book benefits clinicians, educators, investigators, and health administrators. |
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Stranica 40
... synaptic density , infe- rior parietal synaptic density , and inferior parietal neuritic plaques that accounted for 92 % of the variance in test perfor- mance . These results led Terry and colleagues ( 3 ) to suggest that the loss of ...
... synaptic density , infe- rior parietal synaptic density , and inferior parietal neuritic plaques that accounted for 92 % of the variance in test perfor- mance . These results led Terry and colleagues ( 3 ) to suggest that the loss of ...
Stranica 197
... SYNAPTIC ALTERATIONS The major intellectual functions are largely dependent on the patterns of interconnectivity ( see chapter by Hof and Morrison ) and plastic activity in the neocortex . The integrity of these microcircuits depends on ...
... SYNAPTIC ALTERATIONS The major intellectual functions are largely dependent on the patterns of interconnectivity ( see chapter by Hof and Morrison ) and plastic activity in the neocortex . The integrity of these microcircuits depends on ...
Stranica 202
... synapses BnM tangles BnM neurons BnM synapses MF plaques Unnaccounted 50 % 24 % The average concentrations of tangles , plaques , Hirano bodies , granulovacuolar bodies , neuron loss , and synaptic loss are clearly and significantly ...
... synapses BnM tangles BnM neurons BnM synapses MF plaques Unnaccounted 50 % 24 % The average concentrations of tangles , plaques , Hirano bodies , granulovacuolar bodies , neuron loss , and synaptic loss are clearly and significantly ...
Sadržaj
Clinical Presentation | 6 |
Noncognitive Symptoms of Alzheimer Disease | 25 |
Neuropsychology of Alzheimer Disease | 39 |
Autorska prava | |
Broj ostalih dijelova koji nisu prikazani: 26
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abnormal Acad Sci USA activity allele Alzheimer disease Alzheimer's disease amyloid precursor protein Ann Neurol ApoE apolipoprotein apoptosis Arch Neurol areas Aẞ associated atrophy B-amyloid basal forebrain behavioral Biol Brain Res cells cerebral cortex cholinergic cholinergic neurons chromosome clinical cognitive impairment correlation cortical deficits degeneration demented density diagnosis disorders drug effects elderly entorhinal ethnic Exp Neurol familial frontal frontotemporal dementia function gene genetic growth factor hippocampus human immunoreactivity increased lesions levels Lewy body lobe Masliah memory mer's disease metabolism microglia monkeys mutations Natl Acad Sci neocortex neuritic Neurobiol Aging neurodegenerative neurofibrillary tangles neurofilament Neurology neuronal loss neurons neuropsychological Neurosci Neurosci Lett normal onset Parkinson disease pathological patients peptide phosphorylation presenilin prevalence Proc Natl Acad Prpsc Psychiatry receptor regions reported risk factors senile dementia senile plaques studies subcortical symptoms synaptic syndrome tau protein temporal tion transgenic mice trophic factors vascular dementia