Aphasia, Alexia, and AgraphiaChurchill Livingstone, 1979 - Broj stranica: 213 |
Iz unutrašnjosti knjige
Rezultati 1 - 3 od 47.
Stranica 22
... cause of intracranial hemor- rhage . The exact pathology produced by cerebral vascular disorders varies consid- erably . Both thrombosis and embolism cause acute ischemia in the tissues receiving their vascular supply from the occluded ...
... cause of intracranial hemor- rhage . The exact pathology produced by cerebral vascular disorders varies consid- erably . Both thrombosis and embolism cause acute ischemia in the tissues receiving their vascular supply from the occluded ...
Stranica 89
... cause by Kertesz and McCabe ( 1977 ) . Intracerebral hematoma is an uncommon but recognized cause of TCS . The site of pathological lesion in transcortical sensory aphasia is even less exact than that of transcortical motor aphasia ...
... cause by Kertesz and McCabe ( 1977 ) . Intracerebral hematoma is an uncommon but recognized cause of TCS . The site of pathological lesion in transcortical sensory aphasia is even less exact than that of transcortical motor aphasia ...
Stranica 163
... cause of first seizure in persons over the age of 60 ( Adams and Victor , 1977 ) . Our experience in managing sizeable numbers of aphasic patients suggests that these figures may be high , but there can be little doubt that the etiology ...
... cause of first seizure in persons over the age of 60 ( Adams and Victor , 1977 ) . Our experience in managing sizeable numbers of aphasic patients suggests that these figures may be high , but there can be little doubt that the etiology ...
Sadržaj
Introduction | 1 |
Historical Background | 12 |
Neuropathological Substrate of Aphasia | 18 |
Autorska prava | |
Broj ostalih dijelova koji nisu prikazani: 15
Uobičajeni izrazi i fraze
ability abnormality accepted activities additional agraphia alexia anatomical anomia aphasic aphasic patients aphasic syndromes appears approach apraxia associated auditory become Benson better brain Broca aphasia called cause cerebral characteristics clinical combination common complication comprehension conduction aphasia considerable considered consistently correlation cortical damage defect demonstrated described descriptions developed difficulty discussed disorder disturbance dominant evaluation examiner fail field findings fluent frequently frontal hand hemisphere important improvement indicate individual involving language function later lesion less limited localization loss major material motor neuroanatomical neurologic nonfluent normal noted observations occur offer output particularly pathology patient performed posterior present problems produce proved pure recent recognized recovery remains repetition reported scan seen sensory separate severe significant specific speech spoken language studies suggested syndrome techniques term therapy tion transcortical types understand usually variations variety vascular verbal output visual Wernicke aphasia writing written