Aphasia, Alexia, and Agraphia |
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Stranica 18
By necessity then , a neuropathology of aphasia exists and the type of pathological involvement assumes considerable ... contains surprisingly little specific detail about the pathology , and even reports on the neuroanatomical loci of ...
By necessity then , a neuropathology of aphasia exists and the type of pathological involvement assumes considerable ... contains surprisingly little specific detail about the pathology , and even reports on the neuroanatomical loci of ...
Stranica 46
A superior quadrantanopsia strongly suggests temporal lobe pathology ( involving Meyer's loop ) while the presence of an inferior quadrantanopsia usually indicates pathology involving the visual pathways deep in the parietal lobe .
A superior quadrantanopsia strongly suggests temporal lobe pathology ( involving Meyer's loop ) while the presence of an inferior quadrantanopsia usually indicates pathology involving the visual pathways deep in the parietal lobe .
Stranica 94
pathology of language , both as separate entities and as complicating factors for the classic cortical aphasic syndromes . THE APHASIA OF MARIE'S QUADRILATERAL SPACE One variety of subcortical aphasia has a long but controversial ...
pathology of language , both as separate entities and as complicating factors for the classic cortical aphasic syndromes . THE APHASIA OF MARIE'S QUADRILATERAL SPACE One variety of subcortical aphasia has a long but controversial ...
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Sadržaj
Introduction | 1 |
Historical Background | 12 |
Neuropathological Substrate of Aphasia | 18 |
Autorska prava | |
Broj ostalih dijelova koji nisu prikazani: 16
Uobičajeni izrazi i fraze
ability abnormality accepted activities additional agraphia alexia anatomical anomia aphasic aphasic patients aphasic syndromes appears approach apraxia associated auditory become Benson better brain Broca aphasia called cause cerebral characteristics clinical combination common complication comprehension conduction aphasia considerable considered consistently correlation cortical damage defect demonstrated described descriptions developed difficulty discussed disorder disturbance dominant evaluation examiner fail field findings fluent frequently frontal hand hemisphere important improvement indicate individual involving language function later lesion less limited localization loss major material motor neuroanatomical neurologic nonfluent normal noted observations occur offer output particularly pathology patient performed posterior present problems produce proved pure recent recognized recovery remains repetition reported scan seen sensory separate severe significant specific speech spoken language studies suggested syndrome techniques term therapy tion transcortical types understand usually variations variety vascular verbal output visual Wernicke aphasia writing written