Aphasia, Alexia, and AgraphiaChurchill Livingstone, 1979 - Broj stranica: 213 |
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Stranica 46
... visual path- ways deep in the parietal lobe . Unilateral visual inattention often has localizing value also . Thus , unilateral extinction to double simultaneous stimulation af- fecting one quadrant or one visual field often has the ...
... visual path- ways deep in the parietal lobe . Unilateral visual inattention often has localizing value also . Thus , unilateral extinction to double simultaneous stimulation af- fecting one quadrant or one visual field often has the ...
Stranica 161
... VISUAL FIELD DEFECT Visual field defects are common in individuals with aphasia , particularly those whose pathology is posterior . The visual field defect may be of little consequence to the aphasia or its rehabilitation , but in one ...
... VISUAL FIELD DEFECT Visual field defects are common in individuals with aphasia , particularly those whose pathology is posterior . The visual field defect may be of little consequence to the aphasia or its rehabilitation , but in one ...
Stranica 213
... Visual agnosia , 171 Visual communication system ( VIC ) , 184 , 185 Visual field defects , complicating aphasia , 161 Visual - spatial agraphia , 126 Visual symbol system , in aphasia therapy , 184 Wepman test for aphasia , 38 Wernicke ...
... Visual agnosia , 171 Visual communication system ( VIC ) , 184 , 185 Visual field defects , complicating aphasia , 161 Visual - spatial agraphia , 126 Visual symbol system , in aphasia therapy , 184 Wepman test for aphasia , 38 Wernicke ...
Sadržaj
Introduction | 1 |
Historical Background | 12 |
Neuropathological Substrate of Aphasia | 18 |
Autorska prava | |
Broj ostalih dijelova koji nisu prikazani: 15
Uobičajeni izrazi i fraze
ability abnormality accepted activities additional agraphia alexia anatomical anomia aphasic aphasic patients aphasic syndromes appears approach apraxia associated auditory become Benson better brain Broca aphasia called cause cerebral characteristics clinical combination common complication comprehension conduction aphasia considerable considered consistently correlation cortical damage defect demonstrated described descriptions developed difficulty discussed disorder disturbance dominant evaluation examiner fail field findings fluent frequently frontal hand hemisphere important improvement indicate individual involving language function later lesion less limited localization loss major material motor neuroanatomical neurologic nonfluent normal noted observations occur offer output particularly pathology patient performed posterior present problems produce proved pure recent recognized recovery remains repetition reported scan seen sensory separate severe significant specific speech spoken language studies suggested syndrome techniques term therapy tion transcortical types understand usually variations variety vascular verbal output visual Wernicke aphasia writing written